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A Case of Cerebellar Dysfunction After Acute Organotin Poisoning
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Suk Hwan Kim, Cheol In Yoo, Jee Hyun Kwon, Jin Ho Bae, Young Cheol Weon, Yangho Kim
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Korean Journal of Occupational and Environmental Medicine 2009;21(3):289-292. Published online September 30, 2009
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DOI: https://doi.org/10.35371/kjoem.2009.21.3.289
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 Abstract
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- BACKGROUND
We performed a follow-up study of an acute organotin poisoning case on the neurological sequelae that remained after 3 years since diagnosis.
CASE
In the previous study, a 43-year-old male was reported for acute organotin poisoning. After 3 years, the neurological sequelae were studied using neurological examination, cognitive tests, neurobehavioral tests (by CAYSYS 2000(TM)), brain magnetic resonance imaging (MRI), and 18F-fluorodeoxyglucose (18F-FDG) PET/CT. Upon neurological examination and other tests, orientation was improved compared to that 3 years before, but cerebellar ataxia, dysmetria and dysdiadochokinesia remained the same, with little change. Brain MRI showed cerebellar atrophy. 18F-FDG PET/CT revealed mildly decreased metabolic activity in the pons and in both cerebellar hemispheres.
DISCUSSION
This is the first brain MRI study on cerebellar atrophy caused by organotin poisoning. Cerebellar ataxia was confirmed by decreased metabolic activity in 18F-FDG PET/CT.
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Citations
Citations to this article as recorded by  - Magnetic resonance imaging of leukoencephalopathy in amnestic workers exposed to organotin
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Journal of Clinical Neurology.2010; 6(2): 64. CrossRef
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